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Ibodies, primarily SC-1173 and SC9040 [160, 161]. AT1 receptors in the RVLM and NTS showed increased AT1 immunoreactivity, while AT2 receptors showed decreased immunoreactivity. Infusion of Ang II into the brain of rabbits to simulate a heart failure model increased AT1 receptor immunoreactivity in the RVLM [162]. Interestingly, viral transfection of AT2 receptors into the RVLM, which was document
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An N-terminus with small acidic motif (DSpSpEE) that is also found in SIBLING proteins [65]. When solubilized, statherin has a random coil conformation. When bound to HA, it takes on a more alpha-helical structure exposing a bacterial binding site [63], leading to bacterial entrapment. Another example is the role of the IDP, DMP1, in preventing kidney and cardiovascular calcification. The double k
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Veloping these vaccines. Live attenuated vaccine can eventually mutate into aVeloping these vaccines. Live attenuated vaccine can eventually mutate into a more virulent form capable of [5] causing diseases , whereas inactivated or killed vaccines and protein subunit vaccines generally generate weak [6] immune responses often requiring the use of adjuvants . Many live attenuated vaccines are
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Tend to be more difficult and expensive to store and toTend to be more difficult and expensive to store and to distribute, since viability must be maintained, often requiring formulation approaches [7] for stabilization . On the other hand, killed/inactivated vaccines have a number of disadvantages. The major challenge is that since cells are never infected with the live microbe, these vacci
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Larreal-RamirezPagecations and anions [62]; the formation of such chains could also be true of the calcium phosphate prenucleation clusters. 2.2 Pathologic Calcification: Role of IDPs Calcifications occurring in unwanted places ?the aorta, salivary glands and soft tissues, are all associated with IDPs. To illustrate, salivary stones occur when mucins, such as statherin, an un-folded protein [63],